The term YUHA was adopted in 1977 by the European League Against Rheumatism, and in 1983 by the American Association of Rheumatology to designate arthritis in childhood for more than three months.
In 1985, on the recommendation of the III All-Union Congress of Rheumatogens, the widespread term juvenile rheumatoid arthritis was proposed to be divided into two independent diseases – juvenile rheumatoid arthritis and juvenile chronic arthritis. This recommendation is not so much terminological,
how much essential meaning, since the title itself shares the JRA and YUHA. Given the lack of a common nomenclature in our country in this manual, we considered it possible to combine all forms of chronic arthritis, including juvenile rheumatoid arthritis, under the term YUHA. Observations on patients with YUHA show convincingly that by no means in all cases does the child experience a progression of the process and destruction of the arteries, in fact, obligatory for RA. The presence of such “benign” forms of UHA requires a very cautious attitude to the diagnosis and, of course, the tactics of active therapeutic intervention.
Data on the prevalence of Yuha is very controversial. Probably the closest to the truth are the numbers 0.3-0.4 per thousand children. Particularly alarming is the trend towards an increase in cases of UHA. Thus, according to the data of Leningrad, over the past 20 years, the number of observed patients has doubled, similar data are presented for other regions of the Soviet Union and abroad.
In childhood, the highest peak in the incidence rate is 2–6 years old, then up to puberty, the incidence remains approximately at the same level, then a slight increase in the incidence of diseases is observed again. At all ages, girls are more often ill, but this prevalence is most pronounced at the age of 2–4 years, when the ratio of girls to boys is 3: 1, and to a somewhat lesser extent after 10 years (1.5: 1 ratio).
When analyzing premorbid background, attention is drawn to the fact that the disease most often develops in children who were previously considered healthy. In the period preceding the disease, approximately 15% of children can be associated with postponed inflammatory diseases, most often acute acute respiratory disease. In the same number of children, the disease is preceded by trauma, and, trauma as a provoking factor, is more often observed in the case of joint disease. I would like to note that in 5–7% of children YUHA occurs on such provoking factors as vaccination (DTP, tetanus, etc.) and the administration of drugs, most often gamma globulin. In these cases, in most children, the disease immediately begins as a system.
Nearly 20% of patients with UHA include relatives with RA and diffuse connective tissue diseases.
Clinic. Undoubtedly, the main clinical symptom, which unites all quite different in clinical; manifestations of the form of Yuha is arthritis.
As is known, the clinical signs of joint damage are composed of three main characteristics: pain, change in shape, and impaired function.